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Recently a remarkable case report was published about a professional German bodybuilder with a hepatocellular carcinoma (liver tumor). What is really remarkable is the fact that his drugintake (stack) is documented just like his foodintake.
Beside all this interesting information this case study also should be seen as a warning. Side effects with AAS usage do exist, especially when you cycle all year round for many years.
What also struck me is that this particular bodybuilder didn’t include any liverprotector in his stack
Development of Hepatocellular Carcinoma Associated with Anabolic
Androgenic Steroid Abuse in a Young Bodybuilder: A Case Report
Case Report of a 37-year-old male professional bodybuilder with a body height of 180
cm and a body weight of 118 kg.
The patient reported AAS use with different kinds of anabolic substances and a stringent diet for increasing muscle mass. For a period of at least five years he has been consuming the following AASs in a daily medication schedule:
Testosterone propionate, Testosterone phenylpropionate, Testosterone
isocaproate, Testosterone decanoate (Sustanon) 250mg
Trenbolone acetate 75 mg,
5alpha-androstanediol (precursor to DHT) 100 mg,
Boldenone and Methandriol dipropionate (OverDrive) 240 mg,
Stanozolol 100 mg,
Oxandrolone 4 tablets of 10mg,
Letrozole (Femara) 0,065 mg
Oxymetholone 3 tablets of 50 mg or methandienone 10 mg.
Oxymetholone or methandienone was discontinued three weeks before
competition. In addition, he took:
Spironolactone (Aldactone) 100 mg
Mesterolone (Proviron) 25 mg,
Fluoxymesterone (Halotestin) 10 mg four weeks
Torasemide eighteen hours before competition.
Furthermore, a daily intake of amino acid, vitamins, and mineral tablets,
T4 200 μg
Growth Hormones 8 I.E.
The nutritional protocol consisted of six small, protein-rich meals
(chicken breast, fish, protein shakes, salad, vegetable, etc.), a high
caloric and high protein-containing diet to build up muscle mass.
Eight weeks before competition, the meals and consequently the caloric
intake were reduced by 50% to reduce subcutaneous fat.
Torasemide, a diuretic, was taken to achieve a more muscular bodily
contour by reducing extracellular and subcutaneous tissue volume.
http://www.hindawi.com/crim/pathology/2012/195607/
On a sidenote, when RWR stopped producing Drive (and other compound that included MD) I advice some UG Labs (like be Hard Core Labs) to start selling a heavier version of Drive called OverDrive:
http://juicedmuscle.com/jmblog/conte...dipropionate-0
Beside all this interesting information this case study also should be seen as a warning. Side effects with AAS usage do exist, especially when you cycle all year round for many years.
What also struck me is that this particular bodybuilder didn’t include any liverprotector in his stack
Development of Hepatocellular Carcinoma Associated with Anabolic
Androgenic Steroid Abuse in a Young Bodybuilder: A Case Report
Case Report of a 37-year-old male professional bodybuilder with a body height of 180
cm and a body weight of 118 kg.
The patient reported AAS use with different kinds of anabolic substances and a stringent diet for increasing muscle mass. For a period of at least five years he has been consuming the following AASs in a daily medication schedule:
Testosterone propionate, Testosterone phenylpropionate, Testosterone
isocaproate, Testosterone decanoate (Sustanon) 250mg
Trenbolone acetate 75 mg,
5alpha-androstanediol (precursor to DHT) 100 mg,
Boldenone and Methandriol dipropionate (OverDrive) 240 mg,
Stanozolol 100 mg,
Oxandrolone 4 tablets of 10mg,
Letrozole (Femara) 0,065 mg
Oxymetholone 3 tablets of 50 mg or methandienone 10 mg.
Oxymetholone or methandienone was discontinued three weeks before
competition. In addition, he took:
Spironolactone (Aldactone) 100 mg
Mesterolone (Proviron) 25 mg,
Fluoxymesterone (Halotestin) 10 mg four weeks
Torasemide eighteen hours before competition.
Furthermore, a daily intake of amino acid, vitamins, and mineral tablets,
T4 200 μg
Growth Hormones 8 I.E.
The nutritional protocol consisted of six small, protein-rich meals
(chicken breast, fish, protein shakes, salad, vegetable, etc.), a high
caloric and high protein-containing diet to build up muscle mass.
Eight weeks before competition, the meals and consequently the caloric
intake were reduced by 50% to reduce subcutaneous fat.
Torasemide, a diuretic, was taken to achieve a more muscular bodily
contour by reducing extracellular and subcutaneous tissue volume.
http://www.hindawi.com/crim/pathology/2012/195607/
On a sidenote, when RWR stopped producing Drive (and other compound that included MD) I advice some UG Labs (like be Hard Core Labs) to start selling a heavier version of Drive called OverDrive:
http://juicedmuscle.com/jmblog/conte...dipropionate-0
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