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What is the different in Tren

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  • What is the different in Tren

    In laymans terms.
    Tren A. Short acting. needed 5 day or more a week.
    Tren E. Long acting . Once every 5-7 days (E5D).
    Attached Files

  • #2
    Does one have faster results than the other? I plan on running a cycle with some sust 270 in a few months. What's your thoughts?

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    • #3
      Well I think the tren a just get in faster and i think it may get into system faster. I like tren a for shows because it suppose to get in faster

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      • #4
        The side effects worse on the A then the E?
        MODERN DAY GUNSLINGER

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        • #5
          SAME ONE JUST GETS IN (tren a) QUICKER AND YOU HAVE TO TAKE IT MORE OFTEN DUE TO THE HALF LIFE, SO ABOUT THE SAME
          Originally posted by Nephilim View Post
          The side effects worse on the A then the E?

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          • #6
            Many more differences..

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            • #7
              Yes, enenthate esters have a higher weight so you get more bang per buck with ace than you do with en if I remember correctly. Can someone with more knowledge (or a better memory lol) post up something about ester weights and how they efect the mg dosage.

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              • #8
                Chemical Formula

                Molecular Weight

                Mg of Testosterone
                Testosterone (no ester)
                C19 H28 O2 288.4 100mg
                Acetate
                C2 H2 O 42.1 87.26mg
                Propionate
                C3 H4 O 56.1mg 83.72mg
                Enanthate*
                C7 H12 O 112.2mg 71.99mg
                Cypionate
                C8 H4 O 124.2mg 69.90mg
                Decanoate
                C10 H18 O 154.3mg 65.15mg



                Milligrams below are the estimated amount of active hormone per 100mg of hormone and ester.

                Boldenone base: 100mg
                Boldenone acetate: 83mg
                Boldenone Propionate: 80mg
                Boldenone Cypionate: 69mg
                Boldenone Undecylenate: 61mg

                Clostebol Base: 100mg
                Clostebol Acetate: 84mg
                Clostebol Enanthate: 72mg

                Drostanolone Base: 100mg
                Drostanolone Propionate: 80mg
                Drostanolone Enanthate: 71mg

                Methenolone Base: 100mg
                Methenolone Acetate: 82mg
                Methenolone Enanthate: 71mg

                Nandrolone Base: 100mg
                Nandrolone Cypionate: 69mg
                Nandrolone Phenylpropionate: 63mg
                Nandrolone Decanoate: 62mg
                Nandrolone Undecylenate: 60mg
                Nandrolone Laurate: 56mg

                Stenbolone Base: 100mg
                Stenbolone Acetate: 84mg

                Testosterone Base: 100mg
                Testosterone Acetate: 83mg
                Testosterone Propionate: 80mg
                Testosterone Isocaproate: 72mg
                Testosterone Enanthate: 70mg
                Testosterone Cypionate: 69mg
                Testosterone Phenylpropionate: 66mg
                Testosterone Decanoate: 62mg
                Testosterone Undecanoate: 61mg

                Trenbolone Base: 100mg
                Trenbolone Acetate: 83mg
                Trenbolone Enanthate: 68mg
                Trenbolone Hexahydrobenzyl Carbonate: 65mg*
                Trenbolone cyclohexylmethylcarbonate: 65mg*

                Exact calculation:
                Formula (trenbolone only): C18 H22 O2
                Formula (enanthate only): C7 H12 O
                Molecular Weight (trenbolone only): 270.3706
                Molecular Weight (enanthate only): 130.1864

                total weight is: 400.557

                270.3706 / 400.557 = 67.5% actual hormone

                Comment


                • #9
                  Metribolone

                  There are different trenbolone analoques, some of normal strength other more effective, some very very strong

                  Methyl tren:

                  Methyltrenbolone, also known as methyltrienolone, is a 17-alpha alkylated compound with a basic methyl group attached to the 17th carbon on the trenbolone molecule. This tweak causes the steroid to be able to bypass at least the first round of liver degradation. Methyltrienolone is a VERY potent androgen, some several 10's of times stronger than testosterone in that regard. Worth noting about methyltrienolone is that in clinical trials it shows extremely poor binding to androgen receptors. It also causes a extensive increase in a steroid deactivating enzyme that is largely the cause of deactivation of DHT2. From what I can gather about liver toxicity, you would probably do more damage with 10mg of methyltrienolone ED than you would with 50mg Halotestin + 300mg Anadrol + 1gram Dbol per day. The liver toxicity of this compound is off the charts! I have never known anybody who has taken this, and I probably never will, because frankly, anyone who uses this compound will not live long enough to tell me about it. Should you not heed my warning about this one, make sure you have ample amounts of Vitamin B6, Milk Thistle, ALA, and drink 1-2 gallons of Gatorade per day. You will need to use every trick in the book to keep yourself from going into acute liver failure. Say, for arguements sake, that you do not die from taking this, you will most surely end up with prostate problems as this steroid is notorious in the scientific literature for causing abrupt changes in the prostate


                  More info:

                  Brands & Products: Originally produced by Negma, but never approved for production.

                  Characteristics:

                  Methyltrienolone is structurally similar to trenbolone (Parabolan/Finaplix), a well-liked and powerful androgen that does not aromatize to estrogen. The difference is the attachment of a 17-alpha-methyl group for oral activity. So one could refer to methyltrienolone as oral trenbolone. It was first explored quite some time ago by Negma in France, the same company that marketed Parabolan (trenbolone). But the drug was never approved by the French government and was hence never produced. The reason was extreme hepatoxicity. Bill Roberts, the biochemist, once commented that taking methyltrienolone made taking insane doses of anadrol and Halotestin together look mild on the liver. While I was unable to find anything in the literature that describes the extent of the liver toxicity, it's a generally accepted fact. That's also why, to the dissapointment of many, you will never find a commercially marketed methyltrienolone product. Its only sold in bulk to labs and universities for research studies involving androgens.

                  Mainly because (and those who wish it was available will wish so even more now) its such a potent androgen. There is some conflicting information in that regard however. Organic chemist Patrick Arnold, head of LPJ research, once stated that methyltrienolone was the most powerful steroid ever, and that statement has been blown out of proportion and taken on a life of its own. While androgenically a very potent steroid, methyltrienolone is still basically trenbolone with a 17-alpha-methyl group. A group that has the tendency to actually reduce the androgenic potency. So it may actually be somewhat milder than trenbolone, on the contrary to what many pseudo steroid guru's are now claiming after reading Pat Arnold's statement. I can't find any other documented effects of the 17-alpha-alkylation influencing androgen binding in a positive way. It's a potent androgen, with more binding than even DHT2, but the study that claims that is mild at the very best about quantifications, whereas people have used the term 1000 times more powerful than testosterone, which is surely exaggerated.

                  What is interesting is that it seems to show nearly no binding for sex-hormone binding proteins, which makes it a popular choice in androgen receptor studies3, since it will demonstrate equal binding in all tissues regardless of the presence and amount of these proteins. No doubt this plays a role in its supposed binding capacity. In this instance the 17-alpha-alkylation may have played a key role, since it has been demonstrated a multitude of times that 17-alpha-methyl groups decrease the binding for sex-hormone binding proteins as well as most other structures, and due to its triple double bond, trenbolone really didn't bind well to these to begin with.

                  One of the findings made in clinical tests with methyltrienolone was the discovery of high amounts of the DHT-deactivating enzyme 3alpha-hydroxysteroid dehydrogenase in muscle tissue4. Once again proof that God meant to keep us humans weak. Hurray for science. Follow-up studies then went on to show that DHT nonetheless showed similar binding in the prostate, and showing little or no presence of the deactivating enzyme. So God would rather have us all die of prostate cancer than gain a few ounces of muscle. It's a comforting thought, no?

                  What methyltrienolone, despite its amazing capacity, still doesn't overcome are the basic problems with any 19Nor compound. First of all its effects on libido. Methyltrienolone still seems to affect our sex drive in such a potent manner that the dreaded Deca **** (temporary impotence) is a very real threat5. Another is that it still binds almost equipotently to the progesterone receptor3. The latter would be of little concern as long as no circulating estrogen is present since methyltrienolone does not aromatize, but could cause problems such as aggravating water retention and gyno (growth of breast tissue in men) if combined with an aromatizing androgen or an estrogen.

                  While many may wish that an incredibly strong androgenic, non-aromatizing compound as this was available for daily use, its not. And if the indications are true, its probably best. I've warned many people for the toxicity of fluoxymesterone, and everything points to it that methyltrienolone makes fluoxymesterone look like Tums tablets in terms of liver toxicity.

                  Stacking and Use:

                  Obviously this section is mostly useless, as any who would use, let alone stack methyltrienolone for any decent period of time, wouldn't really be around long enough to tell us how well it worked. Ideally one would use it alone, while dieting or for the purpose of gaining lean mass. The androgenic potency is slightly higher than that of trenbolone, so the risk for aggravated hair loss, acne, prostate hypertrophy and deepening of voice is not only realistic, but almost likely. If one were to use it, you would probably have to use every trick in the book to protect your liver and stay alive: Alpha Lipoic Acid, Milk thistle, dessicated liver and Vitamin B6. The blood pressure raise would not be mild either. So something to lower blood pressure is advised as well.

                  Of course the best advice is to refrain from using such a compound, although for 99% of the population that is not a problem, and I would assume that the 1% that does have access would know better.

                  References

                  2 Bonne C, Raynaud JP. Methyltrienolone, a specific ligand for cellular androgen receptors. Steroids 1975 Aug;26(2):227-32

                  3 Dube JY, Tremblay RR, Chapdelaine P. Binding of methyltrienolone to various androgen-dependent and androgen-responsive tissues in four animal species. Horm Res 1976;7(6):333-40

                  4 Tremblay RR, Dube JY, Ho-Kim MA, Lesage R. Determination of rat muscles androgen-receptor complexes with methyltrienolone. Steroids 1977 Feb;29(2):185-95

                  5 Baum MJ, Kingsbury PA, Erskine MS. Failure of the synthetic androgen 17 beta-hydroxy-17 alpha-methyl-estra-4,9,11-triene-3-one (methyltrienolone, R1881) to duplicate the activational effect of testosterone on mating in castrated male rats. J Endocrinol 1987 Apr;113(1):15-20
                  Attached Files

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                  • #10
                    combined and made injectable

                    Underground lab launches methyltrienolone and dimethandrolone stack
                    The outfit goes by the name of HardCore Labs and the product that is causing furore is called MT-DMN. [See the picture below.] MT-DMN is an injectable drug. The active ingredients are a cocktail of the anabolic steroids methyltrenbolone and dimethandrolone.
                    According to William Llewellyn, HardCore Labs is a west European business. Llewellyn is the compiler of Anabolics, an annually updated overview of anabolic steroids available on the black market. He writes about HardCore Labs in

                    his entry on dimethandrolone in Anabolics. You can link through to it from the HardCore Labs website hardcorelabs.org.
                    Clandestine laboratories like Hardcore are the only makers of drugs containing dimethandrolone at the moment. Regular pharmaceutical companies have not yet taken dimethandrolone on board because it has not yet been approved anywhere yet. The test-tube and animal studies that have been published so far, however, all indicate that dimethandrolone [see the molecular structure below ? Ed.] is a really powerful muscle builder.

                    Dimethandrolone is a porcupine anabolic steroid that exceeds the muscle building effect of testosterone by a factor of 136. In addition, dimethandrolone does not affect the prostate and the chance of estrogen side-effects is virtually nil.
                    Through its powerful progesterone effect, dimethandrolone suppresses the body?s own production of hormones and sperm almost entirely. Pharmacists are therefore examining whether it?s possible to put dimethandrolone into a male contraceptive. If dimethandrolone does turn out to have any serious side effects, they are likely to be linked to the recovery of the axis.
                    The usual concentration of dimethandrolone in the illegal drugs is 200 mg per millilitre. According to Llewellyn, injections are needed daily or every other day.
                    HardCore Labs? MT-DMN doesn?t contain dimethandrolone alone.

                    The drug also contains 3 mg of methyltrienolone per millilitre. [Molecular formula here ? Ed.] For years methyltrienolone [aka methyltrenbolone] was the most powerful steroid ever made. But it never made it to the market because in human trials it turned out to have devastating effects on the liver. Users? livers were not only affected very quickly, but after stopping the drug the liver continued to deteriorate.
                    The addition of methyltrienolone increases the risks to the liver, comments Llewellyn. He states that it?s clear that MT-DMN is an incredibly powerful stack. Health-conscious users however won?t start taking MT-DMN, but are more likely to look for a drug containing pure dimethandrolone.
                    Conventional doping tests don?t yet pick up dimethandrolone, Llewellyn concludes, but recent events in Greece show that this is no longer the case for methyltrienolone. [speroforum.com]
                    Sources:
                    hardcorelabs.org.
                    Attached Files

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                    • #11
                      All of these are theories, we have performed some real life testing, a guinea pig injected it after bloodwork. When the cycle was over he did an other bloodwork, only slightly elavated. I'll publish the whole article on the new site. Maybe it came because the mT was made injectable I dunno but all the liverdestroying effects didn't show..

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                      • #12
                        Excellent brilliant post Ronny.

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